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Usually the handbag is fixedly coupled with the clothes, and it cannot be easily detached from the clothes. When the handbag is heavy or the object to be carried is heavy, there is a disadvantage that it is quite uncomfortable for women and girls to carry the handbag with them. For example, when the handbag is used to carry books or a small tool, the user or owner needs to fix the handbag on her back or waist with her hands, so the arm is tired easily. When the handbag is accidentally dropped down, the user will be discomfited, and when it is caught by somebody, the user is embarrassed. Furthermore, the handbag is very likely to open, and the articles in the handbag are scattered, so the user has to search for them one by one when the handbag is opened.
In order to solve the problem of the handbag being fixedly coupled with the clothes, the handbag can be detachably coupled with the clothes through a belt, and a coupling structure of such kind is shown in FIG. 1.
The handbag 10 according to FIG. 1 comprises a main body 11, and a fixing portion 12 for coupling the handbag 10 to the clothes. An opening plate 14 is formed on a side of the main body 11 for carrying articles therein, and a locking mechanism 13 is formed on the fixing portion 12. The locking mechanism 13 includes two levers 131, 132,
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Although the group of exocrine pancreatic cancers is heterogeneous in terms of clinical presentation, histological features, and treatment response, the biology of the majority of pancreatic cancers remains unknown. Most of the information on the genetics of these tumors can be obtained through genomic studies of familial pancreatic cancer. Using a whole-genome approach to genome-wide screens, a few families and sporadic individuals have been identified to have pathogenic mutations in genes involved in three distinct molecular pathways of pancreatic tumorigenesis: the cell cycle (cyclin-dependent kinases, tumor suppressors, proto-oncogenes, and retinoblastoma protein), the WNT pathway (beta-catenin, the wingless proto-oncogene), and mismatch repair. A majority of these molecularly oriented studies have focused on one or two genes, which leaves many questions about the molecular pathogenesis of pancreatic tumors unanswered. The present review summarises current knowledge about the molecular genetics of pancreatic cancers, focusing on the genes involved in the three above-mentioned pathways.104 F.3d 356
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We used 60 healthy volunteers to examine gender differences in propofol-induced sedation, loss of response to verbal command, and effects on cardiovascular function. All volunteers received a slow injection of propofol, 2.5 mg. kg(-1), at a rate of 0.25 mg. kg(-1) minute(-1) until a total dose of 20 mg kg(-1) was achieved. Forearm blood flow was measured by venous occlusion plethysmography, and mean arterial pressure (MAP), heart rate (HR), and oxygen saturation (SpO(2)) were monitored continuously. Propofol elicited a dose-dependent decrease in MAP and HR, and a dose-dependent increase in SpO(2), which did not differ significantly between genders. The median time until loss of response to verbal command was significantly greater in women (33 versus 21 seconds, p = 0.006), and loss of response to verbal command was still present after a median of 40 seconds in this group. Although loss of response to verbal command occurred in 25% of women after 30 seconds, this occurred only in 8.3% of men during the same period. Propofol causes equal decreases in MAP and HR in both genders, suggesting that the delay in loss of response